SynGAP Research Fund dba Cure SYNGAP1 (SRF) is a 501(c)3 tax-exempt organization, and your donation is tax deductible within the guidelines of U.S. law. Please keep your receipt as your official record. We'll email it to you upon successful completion of your donation.
Black Canyon ULTRA 100K Race for a Cure!
The Black Canyon Ultras feature point-to-point 100K & 50K courses along the world-class Black Canyon National Recreation Trail. Join us in supporting Aaron Harding as he runs to raise money for SynGAP Research Fund (SRF) dba cure SYNGAP1. SRF is a global group of families committed to accelerating the science to cure SYNGAP1 & to supporting each other.
Race Date- February 8th
Activity
Frequently asked questions
Is my donation secure?
Yes. Your security and privacy is our highest priority. We use industry-standard SSL technology to keep your information safe and secure.
Do I get a receipt?
Yes, we’ll send a receipt to the email address you provide when donating. The receipt will include your donation details, such as the date, amount, and any beneficiary information.
How is my personal data handled?
Your personal data is processed in accordance with our privacy policy and also in accordance with the privacy policy of Classy, our website partner.
What is SRF?
SynGAP Research Fund (SRF) dba Cure SYNGAP1 is a 501(c)(3) public charity incorporated in 2018. The mission is to improve the quality of life of SYNGAP1 patients through the research and development of treatments, therapies and support systems. Completely family led, SRF is a leading funder of SynGAP research. The founders cover SRF overhead, allowing donations to go toward research and programs to help SYNGAP1 patients.
What are SynGAP1-Related Disorders?
The SYNGAP1 gene is located on Chromosome 6 and is responsible for producing the SYNGAP protein. This protein acts as a regulator in the synapses – where neurons communicate with each other. A variant of the SYNGAP1 gene leads to the gene not producing enough SYNGAP protein. Without the right amount of SYNGAP protein we see an increase in excitability in the synapses making it difficult for neurons to communicate effectively. This leads to many neurological issues seen in SYNGAP1 patients.
